N7-methylguanosine (m7G) is a conserved tRNA modification predominantly located at position 46 in the variable loop of certain tRNAs, enzymatically deposited by the METTL1/WDR4 methyltransferase complex. Arraystar m7G TRAC-Seq maps m7G sites at nucleotide-level accuracy using highly specific chemical reactions, enabling quantitative stoichiometry assessment.
Chemical-based m7G detection without antibody affinity
m7G TRAC-Seq (tRNA reduction and cleavage sequencing) is a chemical method for single-nucleotide resolution profiling of m7G throughout the tRNA transcriptome. Isolated tRNAs are enzymatically demethylated and treated with NaBH₄/aniline to induce reductive cleavage specifically at m7G residues.
The 3′ cleavage fragments are ligated with a sequencing adaptor at their 5′-ends where the precise modification sites are located. The generated cDNA library for each sample is sequenced, and the sequencing data are analyzed for m7G sites and modification levels by cleavage scores.
TRAC-Seq (tRNA reduction and cleavage sequencing) is a chemical method for single-nucleotide resolution profiling of m7G throughout the tRNA transcriptome. It relies on NaBH₄/aniline reductive cleavage at m7G residues, with demethylation controls, to identify m7G sites and modification levels by cleavage scores.
Standard m7G TRAC-Seq project — custom designs and add-on analyses available on request
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Key advantages of Arraystar m7G TRAC-Seq
Precise mapping of m7G sites at nucleotide-level accuracy.
Highly specific chemical reactions, instead of antibody affinity, to eliminate background from non-specific antibody binding and enable quantitative stoichiometry assessment.
Demethylation step using the Arraystar rtStar™ tRNA Pretreatment Kit to overcome tRNA modification barriers that block reverse transcription, allowing efficient sequencing of heavily modified tRNAs.
Simultaneous identification of m7G sites and discovery of modification-associated sequence motifs (e.g. RAGGU motif for m7G tRNAs).
Works together with our tRF&tiRNA sequencing to uncover how m7G modification regulates tRF/tiRNA expression levels.
Detailed bioinformatics analyses with volcano plots, read alignments, and motif analysis for m7G in tRNA biology, diseases, and biomarker applications.
m7G in tRNA biology and disease
N7-methylguanosine (m7G) is a conserved tRNA modification predominantly located at position 46 in the variable loop of certain tRNAs, enzymatically deposited by the METTL1/WDR4 methyltransferase complex. It plays a pivotal role in maintaining tRNA structural integrity through formation of a C13–G22–m7G46 base triple, thereby shielding tRNAs from rapid decay pathways.
Functionally, m7G is indispensable for stem cell self-renewal and proper lineage commitment during embryonic development [1]. Moreover, m7G-modified tRNAs enhance the translation efficiency of codon-enriched mRNAs, particularly those encoding oncoproteins involved in cell cycle progression, proliferation, and growth signaling.
m7G dysregulation, especially with METTL1 overexpression, elevates levels of m7G-modified tRNAs (e.g. Arg-TCT-4-1, Lys-CTT, Val-AAC), driving tumorigenesis in diverse malignancies including leukemia, glioblastoma, cholangiocarcinoma, and lung cancer [2-4].
From total RNA to single-nucleotide m7G maps
RNA quality and quantity assessment before the project proceeds, with small-RNA-retaining purification.
Isolated tRNAs are enzymatically demethylated to remove modification barriers, using the Arraystar rtStar™ tRNA Pretreatment Kit.
NaBH₄/aniline treatment induces reductive cleavage specifically at m7G residues; 3′ cleavage fragments are ligated with a sequencing adaptor at their 5′-ends.
The generated cDNA library for each sample is sequenced.
Sequencing data are analyzed for m7G sites and modification levels by cleavage scores, with motif analysis and differential analysis.
Detailed bioinformatics analyses included in m7G TRAC-Seq
The m7G TRAC-Seq includes detailed bioinformatics analyses to facilitate insights into m7G in tRNA biology, diseases, and biomarker applications.
m7G tRNA modification research across biology and disease
m7G is indispensable for stem cell self-renewal and proper lineage commitment during embryonic development (Mol Cell, 2018).
METTL1 overexpression elevates m7G-modified tRNAs (Arg-TCT-4-1, Lys-CTT, Val-AAC), driving tumorigenesis in leukemia, glioblastoma, cholangiocarcinoma, and lung cancer (Mol Cell, 2021; Mol Ther, 2021).
m7G-modified tRNAs enhance translation efficiency of codon-enriched mRNAs encoding oncoproteins involved in cell cycle progression, proliferation, and growth signaling.
Combines with tRF&tiRNA sequencing to uncover how m7G modification regulates tRF/tiRNA expression levels.
Detailed bioinformatics support m7G studies in tRNA biology, diseases, and biomarker applications.
Official Arraystar sample submission requirements for m7G TRAC-Seq
Common questions about m7G TRAC-Seq
Key References for m7G TRAC-Seq
Arraystar m7G TRAC-Seq combines chemical specificity with quantitative stoichiometry assessment — get a quote and a project timeline tailored to your study.